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Clinician orientation sheet

NM_018489.2:c.7357T>C

Concise orientation only. Dates, normal findings, uncertainty, source boundaries, and longitudinal context remain in the complete profile.

Molecular result and profile context

Report transcript + cDNA
NM_018489.2:c.7357T>C
Report protein consequence
p.Ser2453Pro
Zygosity
Heterozygous
Molecular consequence
Missense
Laboratory classification
Likely pathogenic
Classification source
Original report reviewed
Inheritance
De novo
Inheritance source
Original report reviewed
Relevant additional finding
Additional genetic findings were caregiver-reported; details were not established
Normalized MANE/map notation
NM_018489.3:c.7357T>C · NP_060959.2:p.Ser2453Pro
Variant type
Missense
Sex
Female
Age group
Ages 16–20
Protein position
Amino-acid substitution · aa 2,453

Approved public summary

A girl in the 16–20 age group with the ASH1L missense variant NM_018489.2:c.7357T>C, p.Ser2453Pro, and a complex history shaped by autism, severe sensory-based food restriction, earlier failure to thrive, pica, fasting-associated hypoglycemia with ketosis, cyclic vomiting syndrome, chronic constipation, marked impulsivity and elopement risk, thyroid autoantibodies, and significant premenstrual symptoms. She is also determined, musical, socially interested, and strongly motivated by the people and cultures she cares about. Predictability, concrete communication, longstanding therapeutic relationships, firm boundaries, and a well-matched medication regimen have brought meaningful gains in anxiety and regulation.