LITERATURE · PEER REVIEWED

Loss of histone methyltransferase ASH1L in the developing mouse brain causes autistic-like behaviors

Communications Biology · 2021 · Peer reviewed

COMPLETE CITATION

Citation and identifiers

Gao Y, Duque-Wilckens N, Aljazi MB, et al.. “Loss of histone methyltransferase ASH1L in the developing mouse brain causes autistic-like behaviors.” Communications Biology. 4:756 · doi:10.1038/s42003-021-02282-z.

PubMed ID
34145365

RESEARCH QUESTION

How does developmental neural-lineage loss of Ash1l affect cortical development, myelination, cognition, and behavior in mice?

The neural-lineage mouse deletion links Ash1l loss to several developmental brain and behavioral outcomes, while its broad Nestin-lineage design cannot assign them to one cell class or to humans.

INTERPRETATION BOUNDARY

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What this study supports

He Lab developmental neural-lineage deletion supports effects on cortical development, myelination, cognition, and behavior.

What it cannot establish

Nestin-lineage mouse deletion includes multiple neural cell classes and does not isolate a mature astrocyte, microglial, or human mechanism.

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