TISSUE BIOLOGY · EXPRESSION

ASH1L is broadly expressed—including brain, but not brain-only.

Normal reference atlases identify candidate tissues and cell classes. They do not measure altered dosage, protein function, a patient allele, tissue vulnerability, or clinical effect.

HUMAN TISSUE RNA

All 51 displayed HPA/GTEx consensus values.

Different tissues contain different mixtures of cells, so RNA levels do not rank clinical vulnerability. These values compare transcript abundance—not protein activity, variant dosage, cell vulnerability, or clinical effect. Other HPA tissue, cell, and protein modalities are separate datasets.

REFERENCE METHODSOpen classification, protein, and source context

CLASSIFICATION

Low tissue specificityBroad distribution rather than one organ-enriched RNA pattern

PROTEIN

General nuclear expressionProtein estimates vary by assay; RNA and protein are not interchangeable

SOURCE

HPA/GTEx consensus tissue display (51 values shown)Normal-tissue reference data; not patient-specific and not a measure of tissue vulnerability
Open ASH1L in the Human Protein Atlas ↗

Source: Human Protein Atlas / GTEx consensus tissue data. These are normal-tissue reference measurements, and the source may change over time. Detectable RNA does not show that a person's ASH1L variant has altered that tissue. Field definitions and source notes are included in the downloads below.

Browse all 51 displayed tissue values

Values are source-reported consensus RNA abundance in normal reference tissue. They do not measure protein activity or an ASH1L-related phenotype.

Scroll horizontally to review every column.

All 51 ASH1L values shown from the HPA/GTEx consensus tissue chart.
Organ systemTissueConsensus RNA (nTPM)
Brain & eyeCentral nervous system, choroid plexus, and retinaCerebral cortex26.4
Cerebellum34.5
Basal ganglia23.9
Hypothalamus19.0
Midbrain18.6
Amygdala21.0
Choroid plexus7.5
Hippocampal formation21.0
Spinal cord17.5
Retina24.2
EndocrineHormone-producing organsThyroid gland17.4
Parathyroid gland17.1
Adrenal gland16.2
Pituitary gland17.6
Respiratory & upper digestiveAirway and proximal oral–digestive tissuesLung15.4
Salivary gland17.7
Esophagus16.5
Tongue22.7
Gastrointestinal tractLuminal digestive organsStomach17.1
Duodenum9.2
Small intestine17.1
Colon23.9
Rectum10.8
Liver, gallbladder & pancreasMetabolic, biliary, exocrine, and endocrine compartmentsLiver12.8
Gallbladder8.5
Pancreas16.5
Kidney & urinary tractRenal and bladder tissueKidney13.2
Urinary bladder21.3
Male reproductiveGonadal and reproductive tract tissuesTestis11.0
Epididymis19.6
Seminal vesicle14.5
Prostate17.4
Female reproductive & breastGonadal, reproductive, placental, and breast tissuesVagina18.0
Ovary24.5
Fallopian tube17.7
Endometrium20.7
Cervix19.2
Placenta9.7
Breast18.5
Muscle & vascularCardiac, skeletal, smooth-muscle, and vessel tissueBlood vessel27.4
Heart muscle18.5
Smooth muscle11.9
Skeletal muscle33.7
Connective, adipose & skinBarrier and soft-tissue compartmentsAdipose tissue19.3
Skin20.9
Bone marrow & lymphoidHematopoietic and immune-organ tissuesAppendix8.0
Spleen13.9
Lymph node10.2
Tonsil9.1
Bone marrow11.2
Thymus11.3

CELL-TYPE RESOLUTION

The signal spans neural, epithelial, metabolic, muscle, reproductive, vascular, and immune cells.

Human single-cell and single-nucleus atlases refine the bulk-tissue map. HPA classifies ASH1L as low-specificity across single cells and immune cells, while highlighting adrenal-cortex cells and spermatocytes in its tissue-cell analysis and a neuron-enriched signal in deep visual proteomics.

6 HUMAN CELL-CLASS CONTEXTSOpen the cell-class table and source links

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Human cell-class expression context and the limits of interpretation.
Cell classExamples detectedWhat the atlas can support
Neural & glialBrain-region single-cell types and separate deep-visual-proteomics categoriesThe brain-region atlas includes neurons, astrocytes, Bergmann glia, oligodendroglial lineages, microglia, and vascular cells. Separate DVP categories include neurons, astrocytes/neuropil, and microglia/neuropil; those modalities are not one cell-level measurement.
Airway & epithelialCiliated cells, alveolar type 1 and type 2 cells, keratinocytes, secretory cells, and pancreatic epithelial cellsDetection identifies cell contexts for experiments; it does not establish epithelial disease in ASH1L haploinsufficiency.
Metabolic & renalHepatocytes, pancreatic islets, glomerular cells, proximal and distal tubules, and collecting ductsHuman atlas detection is broad. Direct germline loss-of-function mechanisms in these lineages remain largely untested.
Muscle & vascularCardiomyocytes, skeletal myofibers, capillaries, smooth-muscle cells, and vascular support cellsHuman expression is supported; direct functional evidence is strongest for myoblast fusion in a mouse/cell model.
ReproductiveGranulosa cells, oocytes, spermatocytes, and reproductive-tract cell populationsHPA predicts enrichment in spermatocytes and adrenal-cortex cells. A mouse fetal-ovary overexpression study is a dosage-perturbation model, not a germline loss-of-function result.
ImmuneNeutrophils, macrophages, CD4 and CD8 T cells, B cells, dendritic cells, and other blood lineagesHuman immune-cell RNA has low specificity. Mouse macrophage and T-cell studies supply direct lineage mechanisms, but not a human ASH1L immune phenotype.